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SFN promotes the phosphorylative degradation of -catenin while concurrently suppressing downstream targets including c-Myc and cyclin D1, effectively dismantling the transcriptional program that drives CSC self-renewal (71)
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Although there is now a fairly extensive literature confirming that the intracellular phase is crucial in meningococcal pathogenesis, there are important limitations that this article seeks to highlight
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