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Treatment discontinuation due to side effects occurred in 4.3%, 7.1%, 6.2%, and 2.6% of participants in the 5, 10, 15 mg, and placebo groups, respectively [18]

Multi-omics results show that ALOX15 is the main mediator of phospholipid peroxidation, acting as an ignition point during the ischemia stage, igniting phospholipid oxidation and promoting cell ferroptosis

However, on day 5, the control group exhibited significantly longer escape latency ( P = 0.0116

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Ben Rejeb K, Lefebvre-De Vos D, Le Disquet I, Leprince AS, Bordenave M, Maldiney R et al (2015) Hydrogen peroxide produced by NADPH oxidases increases proline accumulation during salt or mannitol stress in Arabidopsis thaliana