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Physiology of the weight-loss plateau in response to diet restriction, GLP-1 receptor agonism and bariatric surgery

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Exendin-4 significantly increased insulin level and phosphorylation of insulin receptor substrate 1 (IRS-1) in rat hippocampus, and could not change the status of tau phosphorylation without insulin in HT22 neurons, suggesting that insulin is required in reduction of tau hyperphosphorylation by GLP-1R agonists (154)

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Our data add another piece to the puzzle by showing that the GLP-1 signalling in the OB modulates hypothalamic circuits to control pancreatic -cell function