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By introducing the direct cellular transport optimization properties of the GHR peptide variant TB-500, researchers are able to track how the regulation of globular actin (G-actin) into filamentous actin (F-actin) accelerates cellular motility, allowing defensive and structural cells to rapidly populate a wound matrix

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These results are in line with previous findings indicating that inhibition of mitochondrial complex I synergizes with mitochondrial antioxidants in impairing cancer cell fitness, supporting the notion that complex I is a dominant determinant of viability (28)

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