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In contrast, chronic overnutritionparticularly with hyperglycemia and lipid excessincreases mitochondrial electron pressure and ROS generation, rapidly overwhelming glutathione buffering capacity and accelerating cardiometabolic dysfunction

They address different biological needs and, in many cases, work best together

To maintain the intracellular redox balance, GSH is replenished through the reduction of GSSG by glutathione reductase enzyme

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